Vasopressin and Declatimity

Incidentally, the comparison shown in the previous post of profiles of predisposition to orthostatic collapses or “just dizziness” is yet another piece of evidence that a high level of vasopressinergic activity of the brain plays an important role in the formation of the trait of Declatimity, while in the formation of the trait of Questimity - conversely, a low level of this activity plays an important role. This especially concerns the balance between Di and Qi.

Let us recall that vasopressin (“antidiuretic hormone” - another name for it), on the one hand, causes fluid to accumulate in the body, thereby raising blood pressure. This effect of vasopressin is mediated by its action on receptors in the renal tubules; it inhibits the excretion of fluid from the body by the kidneys. On the other hand, it also functions as a neurohormone - vasopressin receptors are present on a great many neurons involved in information processing in the CNS.

What is the difference between dizziness on standing (and the accompanying outflow of blood from the head) and just dizziness? Primarily, it lies in the additional factor of hypovolemia, that is, a small amount of fluid in the body, which is precisely due to a low level of vasopressin. And in the socionic profiles, we see in this case a strong predominance of Questimity over Declatimity.

Given that other, even more direct markers of vasopressin level also clearly correlate with the Declatimity-Questimity balance (such as daily water intake and the frequency with which a person feels the urge to go to the toilet “to pee” - they are higher on average among Questims), there seems to be no doubt left about the important role of vasopressin in ensuring the Declatim properties of the psyche.